![Molecules | Free Full-Text | Computational Drug Design Applied to the Study of Metabotropic Glutamate Receptors | HTML Molecules | Free Full-Text | Computational Drug Design Applied to the Study of Metabotropic Glutamate Receptors | HTML](https://www.mdpi.com/molecules/molecules-24-01098/article_deploy/html/images/molecules-24-01098-g001.png)
Molecules | Free Full-Text | Computational Drug Design Applied to the Study of Metabotropic Glutamate Receptors | HTML
![Combined treatment of menadione and calcitriol increases the antiproliferative effect by promoting oxidative/nitrosative stress, mitochondrial dysfunction, and autophagy in breast cancer MCF-7 cells Combined treatment of menadione and calcitriol increases the antiproliferative effect by promoting oxidative/nitrosative stress, mitochondrial dysfunction, and autophagy in breast cancer MCF-7 cells](https://cdnsciencepub.com/cms/10.1139/cjpp-2019-0585/asset/images/large/cjpp-2019-0585f2.jpeg)
Combined treatment of menadione and calcitriol increases the antiproliferative effect by promoting oxidative/nitrosative stress, mitochondrial dysfunction, and autophagy in breast cancer MCF-7 cells
![Combined treatment of menadione and calcitriol increases the antiproliferative effect by promoting oxidative/nitrosative stress, mitochondrial dysfunction, and autophagy in breast cancer MCF-7 cells Combined treatment of menadione and calcitriol increases the antiproliferative effect by promoting oxidative/nitrosative stress, mitochondrial dysfunction, and autophagy in breast cancer MCF-7 cells](https://cdnsciencepub.com/cms/10.1139/cjpp-2019-0585/asset/images/large/cjpp-2019-0585f5.jpeg)
Combined treatment of menadione and calcitriol increases the antiproliferative effect by promoting oxidative/nitrosative stress, mitochondrial dysfunction, and autophagy in breast cancer MCF-7 cells
![Combined treatment of menadione and calcitriol increases the antiproliferative effect by promoting oxidative/nitrosative stress, mitochondrial dysfunction, and autophagy in breast cancer MCF-7 cells Combined treatment of menadione and calcitriol increases the antiproliferative effect by promoting oxidative/nitrosative stress, mitochondrial dysfunction, and autophagy in breast cancer MCF-7 cells](https://cdnsciencepub.com/cms/10.1139/cjpp-2019-0585/asset/images/large/cjpp-2019-0585f3.jpeg)
Combined treatment of menadione and calcitriol increases the antiproliferative effect by promoting oxidative/nitrosative stress, mitochondrial dysfunction, and autophagy in breast cancer MCF-7 cells
![Combined treatment of menadione and calcitriol increases the antiproliferative effect by promoting oxidative/nitrosative stress, mitochondrial dysfunction, and autophagy in breast cancer MCF-7 cells Combined treatment of menadione and calcitriol increases the antiproliferative effect by promoting oxidative/nitrosative stress, mitochondrial dysfunction, and autophagy in breast cancer MCF-7 cells](https://cdnsciencepub.com/cms/10.1139/cjpp-2019-0585/asset/images/large/cjpp-2019-0585con.jpeg)